Pulmonary Biology in Health and Disease by Edward E. Bittar

By Edward E. Bittar

This new booklet offers an obtainable assessment of the sphere of lung biology and illness aimed toward the graduate or scientific scholar and biomedical researcher. The publication begins via contemplating the anatomy and ultrastructure of the lung and the tracheal and bronchial method, the regulate of respiratory in addition to the basics of pulmonary body structure, fuel alternate and move. this is often through dialogue of the law of acid-base stability, excessive altitude body structure and pathophysiology in addition to workout and the pulmonary approach. Chapters stick with at the immunology of the lung, lung damage, bronchial asthma and emphysema, granulomatous lung ailment, inhalation of poisonous components in addition to illnesses of the small airlines. the ultimate bankruptcy considers present learn into lung transplantation.

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C) Two layers seen in an electron micrograph of rat trachea. (Source: Reproduced with permission from Yoneda, Am. Rev. Resp. Dis. ) 2. Biology of Mammalian Airway Epithelium 25 Mucins are responsible for the peculiar viscoelastic properties of the mucous blanket, allowing its propulsion by the cilia. These large (~500 kD) compounds are bound together in several ways, including hydrogen bonds, electrostatic and hydrophobic interactions, entwinement, disulphide bonds, and lectinlike bonding between carbohydrate and peptide portions of the molecules (Silberberg, 1988).

The human lung starts as a ventral protrusion of the foregut about 4 weeks after fertilization. By 7 weeks the segmental bronchi are formed, and all the bronchi and bronchioles are formed at 16 weeks (Burri, 1985). Gland differentiation spreads centrifugally, with buds appearing at 10–12 weeks of fetal life, and secreting acini at 18 weeks (Bucher and Reid, 1961). H. 7. Schematic representation of airway epithelium during healthy conditions and during cystic fibrosis. Normally, the degree of hydration of the mucus gel is set by the balance between active CI secretion and active Na absorption.

The other major type of ultrastructural change, which is typical of chronic bronchitis induced by smoking, is goblet cell hyperplasia in which the numbers of goblet cells increase at the expense of ciliated cells. , 1983). , 1978). 6. Alterations in mucociliary clearance in chronic bronchitis. (A) Ciliary fields in the trachea of a healthy rat. The small arrows show the direction of mucus transport in each field, and the large arrow points cranially. (B) Ciliary fields in an animal with bronchitis.

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